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Three Simple Ways to Reduce Your Risk of Heart Disease
Ronald Grisanti D.C., D.A.B.C.O., M.S.
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Are you still chasing the cholesterol villain?

Do you really believe that cholesterol is the cause of heart disease?

If you are sold on the theory that cholesterol is the primary cause of heart disease, then you might as well ignore this article.

However, if you have come to the realization that just maybe there are other reasons for heart disease then read on.

Every year, traditional doctors write millions of prescriptions for cholesterol-lowering medications in hopes of preventing heart disease.

How foolish!

These drugs will not improve your health. It's unfortunate what people are going through with statin drugs.

  • Severe fatigue
  • Depression
  • Kidney failure
  • Muscle pain
  • Risks for cancer
  • Suicide
  • Weight gain
  • impotency
  • Death

    They are long-term toxic burdens and they sidetrack doctors and patients from the real cause of our epidemic of heart disease.

    Cholesterol is NOT the cause of heart disease. Instead of reducing cholesterol, you need to reduce inflammation in the arteries.

    You see when you have an injury, whether it's a cut on the skin or irritation to the lining of the arteries, your immune system launches an inflammatory response.

    Now I want you to understand this:

    Long-term inflammation of your arteries will result in large patches of plaque build-up.

    Plaque build-up from inflammation NOT cholesterol
    Plaque build-up from inflammation NOT cholesterol

    At some point, these plaque deposits can dislodge. This can cause a heart attack or a stroke.

    Three Simple Ways to Reduce Inflammation

    1. Reduce Homocysteine. When you have too much homocysteine in your blood, your blood vessels cannot dilate properly. Inadequate blood flow to the heart can cause heart attacks; inadequate blood flow to the brain can cause strokes.

    2. Lower C-Reactive Protien. Elevated CRP levels indicate that there is inflammation in the body. Using this measure, we can detect hidden heart disease better than with cholesterol levels.

    3. Take Care of Your Teeth. Studies show that people with gum disease suffer heart attacks more often than those with healthy gums and teeth.

    New research as found that bacteria in your mouth can sneak through diseased gums and enter the bloodstream causing inflammation. This in turn activates white blood cells, creating arterial lesions leading to a build-up of plaque on the arterial walls.

    Fortunately, you can easily prevent or reverse gum disease. Start by brushing your teeth for three to four minutes at least twice a day.

    Floss once a day, and schedule regular cleanings with your dentist.

    Maintaining healthy gums and teeth will not only save your smile; it may help to save your life.

    The Most Powerful Supplements To Reverse Heart Disease


    Stay Informed and Subscribe to Dr. Grisanti's Free Health Tip of the Week


    References

    Yang F, Tan HM, Wang H. Hyperhomocysteinemia and atherosclerosis. Sheng Li Xue Bao. 2005 Apr 25;57(2):103-14.

    Facila L, Nunez JE, G VB, Sanchis J, Bodi V, Chorro FJ, Llacer A, Chorro FJ. Early determination of homocysteine levels in acute coronary syndromes, is it an independent prognostic factor? Int J Cardiol. 2005 Apr 20;100(2):275-9.

    Sobol AB, Bald E, Loba J. Fractions of total plasma homocysteine in patients with ischemic stroke before the age of 55 years. Angiology. 2005 Mar-Apr;56(2):201-9.

    Church T, Barlow CE, Earnest CP, et al. Association between cardiorespiratory fitness and C-reactive protein in men. Arteriosclerosis and Thrombosis: Journal of Vascular Biology. 2002 Nov 1; 22(11):1869-1879.

    Glurich I, Grossi S, Albini B, Ho A, Shah R, Zeid M, Baumann H, Genco RJ, De Nardin E. Systemic inflammation in cardiovascular and periodontal disease: comparative study. Clin Diagn Lab Immunol. 2002 Mar;9(2):425-32.

    Genco R, Offenbacher S, Beck J. Periodontal disease and cardiovascular disease: epidemiology and possible mechanisms. J Am Dent Assoc. 2002 Jun;133 Suppl:14S-22S.



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