Has Your Doctor Recommended this 'Life-Saving" Vitamin with Coumadin?
Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S.
If you or a loved one has been prescribed the blood thinner Coumadin (warfarin), and have not had your Vitamin K-2
level measured and adjusted for your prothrombin time, you are not receiving the best and most advanced cardiovascular treatment.
Most people are unaware of the fact that coumadin pulls calcium out of bones and dumps it right into the coronary arteries and heart valves.
Worse, taking Coumadin guarantees you will need a stent, bypass surgery or a heart valve replacement sooner than you would if you were not taking this medication.
Coumadin pulls calcium out of bones and dumps it right into the coronary arteries and heart valves.
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And to add insult to injury, it causes high blood pressure, leads to osteoporosis, easy hip fractures, joint replacements, and much more.
But scientific research shows that the right amount of a non-synthetic Vitamin K-2
can stop this from happening. And it does so without damaging the coagulation time.
With the above information deeply ingrained in your conscious, you want to be absolutely sure your cardiologist checks your level of Vitamin K-2
as well as adjust the dose he gives you to the prothrombin time.
But there's an even more urgent reason why you are being grossly cheated if your cardiologist does not have you on the proper dose of vitamin K.
The reason?
Vitamin K2 actually can reverse the damage of aging. It rips calcium off the coronaries and calcified heart valves. It causes regression or melting away of coronary plaque, something no other medicines like the statins (like Lipitor, Vytorin, Crestor, etc.) can claim.
My suggestion?
If your cardiologist does not already have you on Vitamin K-2
, but has prescribed Coumadin, you want to insist on the right treatment.
References:
Schurgers LJ, et al, Regression of warfarin induced medial elastocalcinosis by high intake of vitamin K in rats, Blood 1092823-31, 2007
Gast GCM, et al, A high menaquinone intake reduces the incidence of coronary heart disease, Nutr dletab Cardiorasc Di.s. 2008
Price EA, et al, Warfarin causes rapid calcification of the elastic lamellae in rat arteries and heart valves, Arterioscler Thromb Vase Biol 18:1400-07, 199!
Essahili R, et a1, A new model of isolated systolic hypertension induced by chronic warfarin and K-1 treatment, Am J Hr pert 16:1(13-10, 2003
Spronk HMH„ et al. Tissue-specific utilization of menaquinone-4 results in thi prevention of arterial calcifications in warfarin-treated rats, J Vase Res. 40:531-7, 2003
Geleijnse JM, et al, Dietary intake of menaquinone is associated with a reduced risk of coronary heart disease: the Rotterdam Study, J ;Vuh' 134:310005, 2004
Schurgers LJ, et al, Vitamin K-containing dietary supplements: comparison of synthetic vitamin Kl and natto-derived menaquinone-7, Blood 109:3279-83. 2007
Tanko LB, et al, Relationship between osteoporosis and cardiovascular disease in postmenopausal women, J Bone Min Res 20:1912-20, 2005
Braam LM, Hoeks A, Brouns F, et al, Beneficial effect of vitamin K on the elastic properties of the vessel wall in postmenopausal women: a follow-up study, Thromb Haemost 91:373-80, 2004,
Szulc P, et al, Serum undercarboxylated osteocalcin is a marker of the risk of hip fracture in elderly women, J Clin l mestig 91:1769-74, 1993
Luukinen H, et al, Strong prediction of fractures among older adults by the ratio of carboxylated to total serum osteocalcin, J Bone Min Res 15:2473-78, 2000
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